Senior Lecturer
Medicine
Email : sumeth@med.sab.ac.lk
BSc Hons Biochem (Bom), MPhil Biochem & Mol Biol (Pera), MRes, DIC Bio Med Sc (Imperial Col Lon), DPhil Physiol Anat Genet (Oxon)
Postdoctoral Fellow, National Cancer Institute/NIH, USA
As a translational cancer biologist I am interested in exploring how biochemical pathways and pharmacological interventions determine effective cancer therapies. A snapshot of my research displays how biological questions uncover regulatory mechanisms that control malignant changes in mammalian cells. My particular interest in PI3K-AKT-mTOR regulation of extracellular vesicles called exosomes and hairlike organelles called cilia have shaped my understanding about these powerful signaling platforms in membrane biology of cancer cell-cell communication.
Scholarships and Awards:
Oral and Poster Presentations:
2020 National Postdoctoral Association
2019 American Society for Cell Biology (ASCB) https://www.ascb.org/
2018 Federation for American Society for Experimental Biology (FASEB) https://www.faseb.org/
2014 International Society for Extracellular Vesicles (ISEV) https://www.isev.org/
2012 Life member of the Oxford Union
Nutrient imbalance causes metabolic stress at cellular and organismal level. Diet-induced metabolic dysregulation especially lipid-dependent metabolic stress (LMS) is implicated in tumour initiation and metastasis. In particular, certain fat receptors interact with several ECM molecules including integrins is a key upstream regulator in lipid metabolism and collagen degradation leading to pathogenesis of fat receptor-mediated signaling that disrupts ECM homeostasis.
Oral Submucous Fibrosis (OSF) is a potentially malignant condition with 7-13% transformation rate, which is frequently found in South Asia and in the Western-Pacific. Areca-nut chewing that progressively restricts mouth opening contributes to diet related metabolic stress in OSF with nutritional complications. However, little is known about the malignant transformation of OSF that has undergone epithelial atrophy and submucosal fibrosis with signs of inflammation and immune reactivity. More recently, it was identified that an 80% of malignant transformed cases from OSF showed a significant upregulation of αvβ6 integrins and formed a unique population of keratinocytes with a potential for invasion and metastasis.
The poor dietary habits following chronic impact on restricted mouth opening, OSF patients develop diet-related metabolic stress which we reckon to play a pivotal role in its malignant transformation.